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people are awesome

Almost everything about a human creature is ridiculous, except it’s ability to suffer bravely, and die gallantly, for whatever it loves and believes in.

The validity of that belief, the appropriateness of that love, is irrelevant; it is the bravery and the gallantry that count.

These are uniquely human qualities…

-stone

a moment of silence…

as my 42 minutes of silence (8:46 am – 10:28 am september 11) comes to a close, i am thankful.

this image:

never forget.

says it all for me today.

-stone

long haul wireless with short haul gear

So life has been crazy lately, finding me on the road more often than not, tethering my cell phone whenever I need to get online.

I run an Internet consulting / Infomediary company, so my Internet usage is pretty heavy sometimes, and my network needs are far above what the average end user would be happy with.

I tether as a last resort, because sprint will throttle your connection after you pull the first 5 gigs from the same tower, and in some places (the shop) I’ll hit that pretty quickly…

7 days in fact:

*sigh* I wasn’t even doing anything heavy (no video teleconferencing, streaming, etc.)

I even set windows update not to download patches unless I specifically tell it to. Same thing with antivirus and so on.

Almost 7 gigs in 7 days. :(

So I have a couple of dear friends that live nearby, who I do work for now and again…

It seems they are quite happy to exchange tech support for a connection… :)

Thing is, there’s a lot of distance, a lot of trees, and a railroad between us, so my old “string cat 5 through the culverts” trick isn’t going to work here.

Looks like all the proper hardware to do this “by the book” is about $300 bucks. :(

Hmmm… I used to do a lot of 11 meter (citizens band / cb radio) skip shooting back in the day… so I actually know just enough to be dangerous when it comes to this sorta thing…

Gone are the days of stacked:

and cophased:

(Co-phasing or “stacking” has long been a way to get high gain from antennas. Co-phasing involves placing two (or more!) identical antennas either side-by-side or one over top another (“stacking”) at a certain distance apart (usually a 1/2 Wavelength or more) and feeding the antennas in-phase. The result is 3 db more than just a single antenna.)

Moonraker 6 beams:

60 ft (the legal limit) off the roof of our house on the hilltop outside of Bellingham (during my 11 meter / cb radio days) … but knowledge is knowledge, right? (I had a summer job once :) )

So my awesome neighbor has a Netgear WNR-1000-v2 wireless router…
My end is a Linksys WRT54Gv8 / GSv7 running DD-WRT v24-sp1 firmware…

You know, these:

and for under 10 bucks, my local Wal-Mart supplied these:

That’s a pair of “Rubbermaid 16-Cup Dry Food” containers.

I had intended on using a pair of the “Rubbermaid 1776473 21 Cup Dry Food Storage” units, but I couldn’t find them locally, so I went with the smaller ones.

These will require a bit of hacking, but they’ll have less windage, so it may be a win, right? :)

So the first thing we need to do here is to cut a mounting block for the router to fasten to, that can then be fastened to the container itself. (Then the mounting brackets can screw into the back of it).

Then I cut some holes for the antenna (and then the cables, as I didn’t want the inside/outside seal to be in the working portion of the antenna, so I had to get the router closer to the top of the container)

This router is their end, so the connections are
1) cable to network switch in the house (blue)
2) uplink from cable modem (white)
3) and of course “power”.

Note that this deviates from my standard “router to switch is always hot pink” and “modem to router is always red” standard of cable color coding (the more important it is, the brighter it is, servers are yellow, mailserver is orange, dialup concentrators are blue, internal workstations are green)

Anyway:

The router is currently mounted to the block with a liberal amount of high temperature hot melt glue. I later pulled the unit apart, screwed the bottom plate to the block, and then reassembled the router, as the glue didn’t hold up when I accidentally dropped it :(

With the bigger container, in theory, you can just put everything inside the container and call it a day, but this worked out very well for me, as you’ll see later :)

so now to plug the holes and do some weatherproofing…

That’s a length of 2″ abs drain pipe, cut in half lengthwise, and glued to the container with hot melt glue.

It doesn’t have to be pretty, as small as you can make it is best.
Use a couple scraps for the ends, and let it cool for a bit.

Use a very liberal amount of silicone on the antenna holes:

and then do some more hot melt glue filler work:

Just go slow and let things cool off until you have it watertight and reasonably smooth:

Then screw your mounting brackets to the backside:

Those are 2″ pipe hangers, so I can use 2″ abs drain pipe for my mounting mast.
(note that we changed it for this router later, as a mast was really inconvenient at the other end)

Spray paint it white to make it reject heat a bit, camouflage what’s in it, and make it all professional looking…

and we’re done with that one for now. :)

Now we move on to my end of the equation (a Linksys WRT54G v8):

That sucks. Version 8 of this router doesn’t have removable antennas. that’s kinda shitty of them, I expected this kinda half assed crap on the comcast supplied Netgear, but on a Cisco product? for shame.

Oh well, not my first rodeo. :)

Looks like I need to do a bit of grinding on the case:

So the router can snug up against the top of the housing:

Note that on this one we only need one network uplink (technically didn’t *need* it, but I’d rather not have to go up on the roof an take it down if I have to do anything creative…

This operation should be pretty familiar by now…

So lets go mount them up and see what happens…

The other router is going to be mounted somewhere out there…

On the other side of those trees…

Starting to think I need to replace the safety lens protector on my phone… :(

So here’s what my end looks like. That’s a couple lengths of 2″ abs drain pipe (25 ft total)
The other end is shoved into the ground a little bit, and it’s fastened to the eaves in 2 places (my upper and lower roofs overlap right here) both the antenna’s are equal distance from the other end, as best I can eyeball and estimate it…

So lets power this puppy up and see what we get :)

So far so good! :)

And on to the other end of the equation…

Google earth and my protractor show that my target is about 10 degrees or so off of “parallel to the wall”, so in the above pic my target is almost directly behind me, and since the neighborhood has a policy about this kind of thing, I decided discretion was best… (you have to be *in* the back yard to see this installation at all) so, no mast. Not bad for a power cable, 2 runs of cat 5, and a router in plain sight :)

Not the cleanest installation, but it’ll do for now.
Lets go kick the tires :)

I’ve been spoiled by DD-WRT (the firmware running on the Linksys) it makes life so much easier. You can find it [here]

(every time I have to do anything with an end user router running stock firmware, it makes me cry a little inside… dd-wrt makes me smile :) )

So I head up to the bus, log into the Linksys, and see if it works…

Interesting. So straight out of the gate this works. Both routers can see each other, and are talking ok. Lots of errors, but they all seem to be transmit errors from the Linksys side of things (the Netgear statistics page is next to useless) implying that the Netgear is having some troubles.

The eth1 interface is the connection between the routers, over the long haul hop.

Signal wobbles a bit and it’s really weak, but it’s there. :)

I’m frankly surprised this connection is passing data, I have never seen a signal to noise ratio in the negatives that still works ok. Lets see what we can do about that…

So this is a parabolic antenna template, done by Michael Erskine over at FreeAntennas.com
a larger version is [here]

Instructions are simple:

1) Go peruse Michael’s site [here] (you’ll probably learn something :) )
2) Download the template.
3) Open in your graphic editor of choice and resize as needed. Note that the Galvanized sheet stock I got at lowes is 12″ long, so if you don’t want to cut anything, that’s a good target size.

MAKE SURE YOU KEEP THE SQUARE ON THE DIAGRAM SQUARE!

4) Print the image out on whatever. business card stock or acetate (document protector) works well.
5) Thank Michael for his efforts [here]

So here we go.

Start by transferring your template to your chunk-of-antenna-support. I used a piece of very solid oak plywood, because I have lots of it, and using it means I can let the screws bend the metal for me.

Then I cut it out with the scroll saw. Use whatever tool you have and you’re comfortable with. This part matters, so make it as smooth and accurate as you can get it.

I cut outside the line a little:

Then I sanded up to the line on the disk sander.

Then off to Lowes for the bits I didn’t have lying around…
Namely the cheapest parabolic antenna reflector I could find without cutting up the cookie sheets :)

Since it’s 12″ long, I marked out and drilled 11 holes in it, and started screwing it down.

The screws worked well to make my curve, and this is the part where I learned that I used the wrong sized template. It’s perfect if I trim the edges of the steel off, but they aren’t going to hurt anything by leaving them, so for now they stay (I may revisit this later)

Here’s a shot of the backside, with all the screws in:

Pardon the duct tape, It was raining like only the pacific northwest does, and I need to fiddle the mount.

1) “chunk-of-antenna-support” hits the bulge where the cable cover is.
2) “chunk-of-antenna-support” sits too high on the reflector (the reflector hits the box)
3) reflector is taller than it needs to be.

I’m probably going to make a much bigger reflector and mount the entire router in front of it next time.

Screw it, close enough, lets kick the tires :)

*low whistle* so that 88 cents I spent just proved the parabolic reflector works fantastic :)

Keep in mind that the Netgear is the only one with a reflector on it, and that it hasn’t been aimed, tuned, nothing. This is wild ass guess at it’s best.

Looks like the connection wobbles slightly, but it’s *way* better than it was.

For those of you who are wondering how I turned that Linksys into a Repeater bridge, It goes a little something like this:

Take your Linksys WRT54-whatever, and make sure it will support DD-WRTV24 firmware or higher.
The “supported device” database is [here]

Please note that this is *not* for wired connections between two routers, it is a wireless connection only.

Your PRIMARY router must be in AP mode (default) with DHCP Server enabled.

Your SECONDARY (DD-WRT-whatever) router will be configured as the repeater bridge.

Restore factory defaults on SECONDARY router

Do a proper HARD 30-30-30 Reset on the SECONDARY router.
30/30/30 reset:
This will clear out the NVRAM and set dd-wrt back to default values:
With the unit powered on, press and hold the reset button on back of unit for 30 seconds.
Without releasing the reset button, unplug the unit and hold reset for another 30 seconds.
Plug the unit back in WHILE STILL holding the reset button a final 30 seconds.

Connect to your PRIMARY router, and set it’s LAN IP to 10.1.1.1
Connect to your SECONDARY router, and set it’s LAN IP to 10.1.1.254
Set your computer to a static IP of 10.1.1.253

On your SECONDARY router:

Wireless -> Basic Settings
Physical Interface Section
Wireless Mode: Repeater Bridge
Wireless Network Mode: must match primary router
Wireless Network Name (ssid): must match primary router
Wireless Channel: must match primary router
Wireless ssid Broadcast: Enable
Network Configuration: Bridged
[Save]

Virtual Interfaces Section
Add
Wireless Network Name (ssid): Different from Primary Router

NOTE: You can try using the same ssid but there are many reports of random disconnects and/or no connection if the ssid’s are the same.

If using the same ssid doesn’t work for you, use a different ssid from the primary router.

Wireless ssid Broadcast : Enable
AP Isolation : Disable
Network Configuration : Bridged
[Save]
Wireless -> Wireless Security
Physical Interface Section
Security Mode : must match primary router

(should be WPA2-AES, as WEP can be cracked in seconds by anyone marginally competent).

WPA Algorithms : must match primary router
WPA Shared Key : must match primary router
Key Renewal Interval (in seconds) : Leave default

Virtual Interfaces Section
(note if you don’t see this section your firmware should be at least v24-sp2)

Security Mode : must match physical interface
WPA Algorithms : must match physical interface
WPA Shared Key : must match physical interface
Key Renewal Interval (in seconds) : Leave default
[Save]

Setup -> Basic Setup
Connection Type will be: Disabled
Set STP for Disabled (Enabled sometimes can cause connection problems)
IP Address : 10.1.1.2 (Assuming Primary Router IP is 10.1.1.1)
Mask : 255.255.255.0
Gateway: 10.1.1.1 (again assuming Primary Router IP is 10.1.1.1)
DHCP Server: Disable
Local DNS: 10.1.1.1 (if IP of Primary Router is 10.1.1.1)
Assign WAN Port to Switch : Optionally enable this to use the WAN port as another LAN port.
[Save]

Setup -> Advanced Routing
Set Operating mode to “Router”
[Save]

Open Services
Disable Dnsmasq
[Save]

Security -> Firewall tab
Uncheck all boxes, except Filter Multicast
Disable SPI firewall
[APPLY]

[Reboot]

Once you have it working, go to the wireless security tab, and set the same type of security AND key for both the primary and the repeater ssids and hit apply.

set your computer back to auto IP and auto DNS if you wish.
(I use 8.8.8.8 and 8.8.4.4 personally)

Your SECONDARY router should now be able to connect wired and wireless clients.

They will receive IP Addresses from the Primary Router.

NOTE: All repeaters, (including this one), will sacrifice half of the bandwidth available from the primary router for clients wirelessly connected to the repeater.

This is a result of the repeater taking turns talking to not just one partner, but to two, and having to relay the traffic between them.

As long as your internet bandwidth requirements are within this halved bandwidth amount there will be little or no reduction in “speed”.

If it doesn’t work properly the first time, check all your steps / settings again, and reboot the PRIMARY router, then the SECONDARY after the PRIMARY comes back up.

So, I finished this project weeks ago, and held off the blog post until I put the parabolic antennas on the Linksys… which I have yet to do because it’s working well enough for now, so here ya go :)

-stone

ups hacking for fun and extended runtime

An uninterruptible power supply, (UPS for short) is an electrical apparatus that provides emergency power to a load when the input power source, (typically the utility mains), fails.

A UPS differs from an auxiliary or emergency power system or standby generator in that it will provide instantaneous or near – instantaneous protection from input power interruptions by means of one or more attached batteries and associated electronic circuitry for low power users, and or by means of diesel generators and flywheels for high power users.

The on-battery runtime of most uninterruptible power sources is relatively short. 5 to 15 minutes being typical for smaller units, but sufficient to allow time to bring an auxiliary power source on line, or to properly shut down the protected equipment.

Today I decided to change that, since I need to be able to run off batteries for a lot longer. :)

As may of you know, I am planning on going on the road for an extended period of time with the mighty warbusaur, and I will most likely be spending my summers in the pacific northwest, and my winters somewhere warmer… :)

So of course I have an epic plan involving solar panels, and wind/tidal/current generation techniques as well.

That stuff is all coming. but in the meantime, as I give the warbusaur (my pd4501 greyhound super scenicruiser) much needed love, I find that I need to run a large number of electronics when there is no power to do so…

I have a generator, but running a generator (even my super awesome honda EU3000is) just to charge my laptop and cell phone, with fuel being $3.95/gal right now, is frankly retarded…

So, how to store power for later usage?

I figured I needed something like 12 hours of runtime, but more is always better (go big or go home) and easier on the batteries, since they won’t discharge as far (deep discharges are what kill batteries)

Hmmm. Batteries.

I know I have some deep cycle trolling motor batteries in the van, but lets see if we can solve a couple problems at once, since batteries don’t like to sit, and my bus has a pair of really big (1400cca!) batteries in it, that are not being maintained properly.

so, I’ll pull them out of the warbusaur, and temporarily use them for this. :)

That my friends is a pair of Interstate Workaholic 1400cca 8d batteries…

Amps: 1750
Cranking Amps: 1750
Cold Cranking Amps: 1400
Voltage: 12
Weight: 130
Width: 11
Length: 20.75
Height: 9.63

They are heavy (260 lbs!), and they aren’t deep cycle, but they are as industrial as they come, and will work fine for what we are doing here. :)

So after much herculean effort, I managed to pull the batteries, and get them on my trusty cart, and drag them the 150 feet or so to the front of the shop.

A bit of work with a terminal shaper, and a scrub brush and some water, and they look as good as new. :)
Well, except for that…

In the depths of time there is a story about argyle pushing a 55,000 lb bus by himself…

We were on our way back from Pennsic, and overnighted in a rest stop up in the rocky mountains.

We awoke to 35 degrees outside, and the warbusaur won’t start. She turns over fine, but that 8v-71 jimmy diesel is just not having any of it.

We were on flat ground, so I dropped it in gear, let out the clutch, and bumped it with the starter, the idea being that if I can get her to move a little, we can just roll down the mountain and pop start it on the exit road from the rest area…

Well, when you walk a 55,000 lb scenicruiser about 50 feet on the starter, it creates a whole lot of current draw, and that in turn creates heat…

The above is what happens when your battery cables get hot enough to melt their jackets off and melt into the battery case…

I can see the plates through the holes on one of these, so I need to do something about it…

Duct tape is tacky, so I got out my trusty hot melt glue gun and did a bit of plastic welding…

Hot melt glue is awesome stuff when you need to fill gaps and stick things together permanently. I am constantly using it for things.

This is the bigger of the two holes in the casing. you can actually see the lead plates through this one.

5 minutes later, the patch is done, and cooling down. after it cooled down I used a utility knife blade and shaved the patch down so it was flush with the rest of the case.

Topped the cells up with distilled water, and put them on my 10 amp slow charger to see if they will take a charge (they should, but they are completely dead, so it may take a bit of cajoling)

So now comes the fun part :)
I can charge these with a battery charger, and discharge them with a small power inverter, and it will work just fine, and the losses are acceptable, but the only inverter I have here isn’t big enough to run my workstation, and it’s a pretty long drive to go fetch it.

But I have a lot of random bits floating around the shop, and we may be able to come up with something a bit more… creative. :)

This is one of those times I wish my solar panels were here, instead of sitting in Arizona waiting for me to buy them :)

So I rummaged around the shop and came up with a pile of dead computer ups systems.

Most dead ups systems aren’t actually dead. usually they work just fine, but the batteries are hosed.

Most of them will have smart chargers in them, and handle the failover from mains (shore) power to battery backup, automagically, as well as do unattended shutdowns of attached equipment (with a bit of creativity).

(although for this instance, I only need that if I’m running my workstation)

So, lets see what we’ve got, shall we? :)

+—-=< \>=—-=< ->=—-==—-=< |>=—-=< \>=—-=< ->=—-==—-+

Exhibit A: Powerware Prestige 1250p2
Google says:
True on-line, double-conversion, powerline isolated, with Cell Saver Technology™ (charges batteries only when necessary, resulting in up to 50% longer battery life)

Capacity
1250VA, 875 Watts
Runtime at full load (875 Watts) 5.2 minutes
Runtime at half load (437 Watts) 14.6 minutes

Looks promising so far…
(giggles to himself at those runtime numbers)

*blink*
now that is interesting.
5 gell cells in series? 5 x 12v = 60 volt dc rails.

Sad day, this won’t work for our needs, because I don’t have 5 batteries.

Interesting design though, looks to be pretty well thought out. Notice the lack of a big transformer?

Upon further inspection, if I had 5 batteries this unit could probably handle it.
There is lots of good stuff in here, it looks like this thing is built out of high end parts and it’s done right, and the cooling design is excellent (which matters a whole lot when you’re going to put it under the kind of stresses that (5) 1400cca 8d’s will put it under).

Alas, I had to put it back on the “I’ll use it sometime” pile. We need something with a 12 or 24 volt rail.

+—-=< \>=—-=< ->=—-==—-=< |>=—-=< \>=—-=< ->=—-==—-+

Exhibit B: Smart Power Systems SS1250RM
Google says:
Fully digitized microprocessor controlled, AVR boost, Advanced battery management, Cold start (DC power on), Automatically charging when UPS off.

That cold start ability is handy (although most ups’s, you can do the same thing by plugging them into themselves if you have to)

Capacity:
1250VA / 750 Watts.
Voltage (on battery) Pure sine wave at 120V ±5% -10% of nominal after low battery warning
Voltage Regulation (AVR)
AVR automatically increases output voltage 15% above input voltage if -9% to -25% of nominal.
AVR decreases output voltage 13% below input voltage if +9% to +25% of nominal.
Transfer Time 2-4 milli seconds, including detection time
Typical Recharge Time 4 hours (to 90% of full capacity)
Run Time (minutes) Half Load 32.4

Nice. AVR is good, but most ups’s have that, (exhibit a does, because the output can be greater than the input for short periods according to the spec sheet)

The run time and recharge numbers are nice, but for my purposes, I would expect to be drained down to more like 70%, and our batteries are so much bigger that it’s meaningless to us (except when comparing to other units)

This is worth a look…

Hmmm… paydirt! :) 24 volt rail… (2) 12 volt batteries wired in series, then wired in parallel with another pair. (series you add the voltages together, parallel the voltage remains the same) so we’re good to go…

Note the large transformer.

The charging circuits on this unit seem to reflect that aforementioned recharge time.
Keep in mind that these systems are designed to slowly charge up over time, not to do it every day.

The charging circuits here look pretty whimpy, especially compared to Exhibit A, and they may die if we put a real battery bank on it, but this unit will do the job.

Lets keep looking…

+—-=< \>=—-=< ->=—-==—-=< |>=—-=< \>=—-=< ->=—-==—-+

Exhibit C: American Power Conversion Smart-ups 650)
Google says:
Capacity:
650VA / 410 Watts
Typical recharge time 6 hour(s)

This is a small, low end unit, but I know people that have used a car battery with one, so lets check it out.

12 volt rails, so we can use 1 battery, or 2 in parallel. This appears to be a pretty robust unit.

410 Watts is enough for my cell phone and laptop, but it won’t run my workstation.
I may put this on a car battery to run my laptop and cell charger, since this unit is a bit more efficient than the big ones (no cooling fans)

This unit works fine, but apparently apc decided to change their powerchute software and remove the old versions from the internet, because I can’t talk to the stupid thing to turn off the “hey!, I’m on battery power!” alarm.

Setting it aside, Wrong tool for the job.

+—-=< \>=—-=< ->=—-==—-=< |>=—-=< \>=—-=< ->=—-==—-+

Exhibit D: American Power Conversion SU3000RM
Google says:
Capacity:
3000 VA / 2250 Watts
Typical recharge time 3 hour(s)

Let’s pop the top and see what we have here..

This just makes me smile.
Big transformers, beefy construction throughout.

This will do :)

Errr. or not. 8 batteries in 2 series parallel banks of 24 volts each, for a 48 volt dc rail.

so i need to buy 2 more batteries, or move on. :(

The temptation to buy 2 more batteries is very very high… :)

I mean, just look at those transformers :)

Woot! it’s even got the smartslot card in it. :)

And that brings us to our last contestant…

+—-=< \>=—-=< ->=—-==—-=< |>=—-=< \>=—-=< ->=—-==—-+

Exhibit E: American Power Conversion SU1400RM
Google Says:
Capacity:
1400 VA / 950 Watts
Typical recharge time 3 hour(s)

This is a similar design to the last unit we looked at, so here’s hoping :)

Pull the faceplate, Pull the battery Plug out…

Pull out the battery drawer…
If this unit is done like most 4 battery apc units are, we may be in luck :)

60 amp fuses on the dc side of things, there are two of them, so that means series parallel :)

And this is what series parallel 24 volt rail wiring looks like :)

Like the other apc units we’ve seen so far, the build quality and the layout are well thought out. I think we have a keeper :)

Looking good so far :)

Nice, well thought out parts layout and cooling…

Mock it up for testing…

So far so good…

You can plug a lot of things into one of these. it’ll run your microwave or your coffee pot all day :)

(The 3,000 va one will do both at the same time all day)

Now I’m going to leave the ups unit charging the batteries all night and see if it blows up or not… :)
-stone

in the round (it’s not just for def leppard anymore)

Little darling, it’s been a long cold lonely winter,
Little darling, it feels like years since it’s been here,
Here comes the sun, here comes the sun,
and I say it’s all right…

The chance to hit the road is always one that I look forward to, and I very rarely turn down a road trip.
These past two weeks were no different, as I engaged in 3412 miles of white line therapy.

Heading East, then South, Sunrise found me ticking off the miles on my way to the city of sin to meet up with my uncle and his lady for some door banging fender rubbing NASCAR craziness :)

…and then I head a bit further south to pick up 1500 lbs of high tech old school engineering…

When my bus is done, I’m seriously going to start spending winters in Vegas and Arizona, because I’ll tell you, 70 degrees in the sun beats the crap out of the “it’s rained for 3 weeks straight and hasn’t gotten above 45 degrees in a month” that we deal with here for 6 months out of the year here in Washington…

I never back down from a weird parking challenge…

although I really detest backing up small trailers :(

This stuff is pretty heavy…
Heavy enough that I wasn’t about to carry it further than I had to.

Packing round things in square spaces is always a bit of a pain, so I got a bit creative :)

At below 55, aerodynamics don’t have nearly the effect on fuel economy that they do at high speeds, and while I figured it was going to be a 55mph slog home regardless, parts of this load needed to be “not flat”.

Add a very heavy chunk of cast iron and a couple heavy bags to the mix, and it made for an interesting game of tetris :)

and everyone needs a UFO strapped to the top of their trailer :)

The sunset as I crossed the Mojave desert on the way home was absolutely gorgeous :)

I suspect the pucker factor moment involving how low the fuel gauge will go, and what happens when the fuel economy computer tells you that you have “—” miles before you are stranded by the side of the road deserves a mention :)

We hit the rain just after grant’s pass, and it washed off the desert dust quite nicely…

No load shift anywhere, my +10 sanguine shades of Tetris are truly appreciated :)

Of course, it all got soaked, and the internal package padding is cardboard, so I’m going to have to unwrap it…

And this is what an unpacked, 20 foot in diameter yurt from [Pacific Yurts] looks like… The craftsmanship is top notch.

What is a yurt?

well…
(thank you wikipedia)
A yurt (üi or kiz üi in Kazakh, ger in Mongolian) is a portable, felt-covered, wood lattice-framed dwelling structure traditionally used by Turkic and Mongolian nomads in the steppes of Central Asia. A yurt is more home-like than a tent in shape and build, with thicker walls.

Etymology and synonyms
The word yurt is originally from a Turkic word referring to the imprint left in the ground by a moved yurt, and by extension, sometimes a person’s homeland, kinsmen, or feudal appanage. The term came to be used in reference to the physical tent-like dwellings only in other languages. In modern Turkish the word “yurt” is used as the synonym of homeland. In Russian the structure is called “yurta” (юрта), whence the word came into English.

The Kazakh word used for yurt is киіз үй (transliterated: kïiz üy), and means “felt house”. The Kyrgyz term is боз үй (transliterated: boz üy), meaning “grey house”, because of the color of the felt. In Turkmen the term is both ak öý and gara öý, literally “white house” and “black house”, depending on its luxury and elegance. In Mongolian it is called a ger (гэр / ᠭᠡᠷ᠌). Afghans call them “Kherga”/”Jirga”. In Pakistan it is also known as gher (گھر). In Hindi, it is called ghar (घर), which means home. In Persian yurt is called xeyme (خیمه), in Tajik the names are yurt, xona-i siyoh, xayma (юрт, хонаи сиёҳ, хайма).

Construction
Traditional yurts consist of a circular wooden frame carrying a felt cover. The felt is made from the wool of the flocks of sheep that accompany the pastoralists. The timber to make the external structure is not to be found on the treeless steppes, and must be obtained by trade in the valleys below.

The frame consists of one or more lattice wall-sections, a door-frame, roof poles and a crown. Some styles of yurt have one or more columns to support the crown. The (self-supporting) wood frame is covered with pieces of felt. Depending on availability, the felt is additionally covered with canvas and/or sun-covers. The frame is held together with one or more ropes or ribbons. The structure is kept under compression by the weight of the covers, sometimes supplemented by a heavy weight hung from the center of the roof. They vary regionally, with straight or bent roof-poles, different sizes, and relative weight.

A yurt is designed to be dismantled and the parts carried on camels or yaks to be rebuilt on another site.

Enthusiasts in other countries have taken the visual idea of the yurt—a round, semi-permanent tent—and have adapted it to their cultural needs. Although those structures may be copied to some extent from the originals found in Central Asia, they often have some different features in their design that adapt them to different climate and use.

In the United States and Canada, yurts are made using hi-tech materials. They are highly engineered and built for extreme weather conditions. In addition, erecting one can take days and they are not intended to be moved often. These North American yurts are better named yurt derivations, as they are no longer round felt homes that are easy to mount, dismount and transport. North American yurts and yurt derivations were pioneered by William Coperthwaite in the 1960s, after he was inspired to build them by an article about Supreme Court Justice William O. Douglas’ visit to Mongolia.

In 1978, Oregon-based company Pacific Yurts became the first to manufacture yurts using architectural fabrics and structural engineering, paving the way for yurts to become popular attractions at ski resorts and campgrounds. In 1993, Oregon became the first state to incorporate yurts into its Parks Department as year round camping facilities. Since then, at least 17 other US States have introduced yurt camping into their own parks departments.

In Europe, a closer approximation to the Mongolian and Central Asian yurt is in production in several countries. These tents use local hardwood, and often are adapted for a wetter climate with steeper roof profiles and waterproof canvas. In essence they are yurts, but some lack the felt cover that is present in traditional yurt.

Different groups and individuals use yurts for a variety of purposes, from full-time housing to school rooms. In some provincial parks in Canada, and state parks in several US states, permanent yurts are available for camping.

Neat huh? :)

So we hit Vegas again on the way back… and I kinda regret coming home. It’s warm there :)
-stone

phoenix rising…

I’m beautiful in my way, ’cause god makes no mistakes, I’m on the right track, baby I was born this way…

-stone

back on track

Since my mother’s heart attack in march of 2010, and my adventure a day after they released her, I’ve been trying to keep my act together health wise.

I’ve been doing a 5 mile fast walk most days, with the intent of keeping my heart rate up for at least 45 minutes, as I try to keep in compliance with the 200 minutes of exercise a week (or better) rule.

I’ve got a nice route I do, lots of things to see and quite varied terrain.

The first mile of it is all downhill, (can’t be helped because of where I live) which sounds nice and easy, but that means the last mile is all uphill. :(

To be honest, I miss my roller skates. I’ve considered getting outdoor wheels for them, but quads and Washington weather don’t really work well together.

Generally it’s been working. I’ve still got a sysadmin belly, which I blame on fast food and soda, but I’ve got my wind back and I physically feel better.

I’ve had a lot of major upheavals in my life recently, completely derailing the balance of health, wealth, and relationships, that one needs to have to be happy.

The past 8 months have been very hard, mostly due to events beyond my control, and the sheer amount of stress that I’ve been dealing with for the past couple years.

Major Depression, lack of sleep, inability to focus, just to name a few.

Considering who I am and the kind of things I do, this is a really bad sign.

I was taking these supplements:

 

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Spring Valley, Enteric Coated, Fish Oil. (3600mg a day, 1080mg a day of omega 3)

Fish oil has been studied for treating clinical depression, anxiety, and high blood pressure.

Countries with the highest intake of fish in their diets are correlated with the lowest rates of depression among citizens.

Several studies report possible anti-cancer effects of n−3 fatty acids found in fish oil (particularly breast, colon and prostate cancer).

Omega-3 fatty acids reduced prostate cancer growth, slowed histopathological progression, and increased survival in genetically engineered mice.

Among n-3 fatty acids (omega-3), neither long-chain nor short-chain forms were consistently associated with breast cancer risk.

High levels of docosahexaenoic acid, however, the most abundant n-3 polyunsaturated fatty acid (omega-3) in erythrocyte membranes, were associated with a reduced risk of breast cancer.

In a recent study of 35,000 middle-aged women published in the journal Cancer Epidemiology, those that took fish oil supplements were found to have a 32% lower risk of breast cancer, although the study authors stressed that the result was preliminary and that “we should not draw any conclusions about a causal relationship.”

The US National Institutes of Health lists three conditions for which fish oil and other omega-3 sources are most highly recommended: hypertriglyceridemia, secondary cardiovascular disease prevention and high blood pressure. It then lists 27 other conditions for which there is less evidence. It also lists possible safety concerns: “Intake of 3 grams per day or greater of omega-3 fatty acids may increase the risk of bleeding, although there is little evidence of significant bleeding risk at lower doses. Very large intakes of fish oil/omega-3 fatty acids may increase the risk of hemorrhagic (bleeding) stroke.”

Studies published in 2004 and 2009 have suggested that fish oil may reduce the risk of depression and suicide. One such study took blood samples of 100 suicide-attempt patients and compared the blood samples to those of controls and found that levels of Eicosapentaenoic acid were significantly lower in the washed red blood cells of the suicide-attempt patients. A small American trial, published in 2009, suggests that E-EPA, as monotherapy, might treat major depressive disorder, however the study achieved no statistical significance.

According to a study from Louisiana State University in September 2005, fish oil may help protect the brain from cognitive problems associated with Alzheimer’s disease.

A study examining whether omega-3 exerts neuroprotective action in Parkinson’s disease found that it did, using an experimental model, exhibit a protective effect (much like it did for Alzheimer’s disease as well). The scientists exposed mice to either a control or a high omega-3 diet from two to twelve months of age and then treated them with a neurotoxin commonly used as an experimental model for Parkinson’s. The scientists found that high doses of omega-3 given to the experimental group completely prevented the neurotoxin-induced decrease of dopamine that ordinarily occurs. Since Parkinson’s is a disease caused by disruption of the dopamine system, this protective effect exhibited could show promise for future research in the prevention of Parkinson’s disease.

Evidence regarding the efficacy of fish oil supplements as a treatment for depression is currently inconclusive. Whereas several methodologically rigorous studies have reported statistically significant positive effects in the treatment of depressed patients, other studies have found effects to be small or insignificant.

For example, an August 2003 double-blind placebo-controlled study published in the journal European Neuropsychopharmacology found that among 28 patients with major depressive disorder, “patients in the omega-3 PUFA group had a significantly decreased score on the 21-item Hamilton Rating Scale for Depression than those in the placebo group.” Another study in the American Journal of Psychiatry reported that the addition of fish oil supplements to regular maintenance anti-depression therapy conferred “highly significant” benefits by the third week of the trial.

Despite the conflicting scientific evidence for the efficacy of fish oil in treating depression, web searches reveal a large amount of anecdotal evidence for its alleged benefits. Notably, it does not appear that any peer-reviewed studies have reported negative effects on depressive symptoms as a result of fish oil supplementation.

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and :

 

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Spring Valley High Potency B Complex (one per day, in the morning)

Promotes energy and metabolism.

Recommended as part of daily regimen for health, stress, mood, and energy.

Vitamin B-6 builds body proteins; riboflavin and niacin regulate antioxidant activity.

Specially formulated to replenish the nutrients lost during high stress times.

Vitamin B-1 (Thiamin Mononitrate) 50mg
Vitamin B-2 (Riboflavin) 12.5mg
Vitamin B-3 (Niacinamide) 50mg
Vitamin B-6 (Pyridoxine Hydrochloride) 12.5mg
Folate (Folic Acid) 50mcg
Vitamin B-12 (Cyanocobalamin) 12.5mcg
Biotin 25mcg
Pantothenic Acid (d-Calcium Pantothenate) 50mg
Calcium (Dicalcium Phosphate) 118mg

The Vitamin B Complex comprises of the essential B Vitamins – Vitamin B1, Vitamin B2, Vitamin B3, Vitamin B5, Vitamin B6, Vitamin B9, Vitamin B12 plus the vitamins Biotin, Choline and Inositol. Vitamin B Complex is needed for the proper functioning of almost every process in the body.

Energy Production
Vitamin B1 is needed to help convert the carbohydrates we eat into glucose. The following B Vitamins are needed at a cellular level to convert glucose into energy – Vitamin B2, Vitamin B3, Vitamin B5, Vitamin B6 and Biotin. A Vitamin B deficiency in any of these vitamins can lead to decreased energy production, lethargy and fatigue.

Healthy Nervous system
The Vitamin B Complex is essential for the healthy functioning of the nervous system. Vitamin B5 is needed for the correct functioning of the adrenal glands and the production of some hormones and nerve regulating substances. Vitamin B1, Vitamin B6 and Vitamin B12 are essential for the regulation and correct functioning of the entire nervous
system including brain function. Vitamin B9 is essential to prevent neural tube defects to the foetus during pregnancy. A deficiency in any of the Vitamin B Complex vitamins can lead to feeling stressed, anxious and depressed.

Good Digestion
The Vitamin B Complex is essential for correct digestion, production of HCl (Hydrochloric acid) and to assist in the breakdown of fats, proteins and carbohydrates. Especially vital for good digestion are Vitamin B1, Vitamin B2, Vitamin B3 and Vitamin B6. A deficiency in any of these B Vitamins can lead to impaired digestion and deficiency of essential nutrients.

Healthy Skin, Hair and Nails
The Vitamin B Complex is essential for correct RNA and DNA synthesis and cell reproduction. As our Skin, Hair and Nails are constantly growing and renewing themselves we need the following B vitamins to ensure the good health of these structures – Vitamin B1, Vitamin B2, Vitamin B3, Vitamin B5, Vitamin B9, Vitamin B12, Biotin and Choline. Deficiencies of any of these B Vitamins can lead to dry, grey skin, dermatitis, wrinkles, acne, rashes, falling hair and weak, splitting nails.

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They were working pretty well for me, so I started taking them when I climbed back in the saddle so to speak.

Thing is, I couldn’t break the depression cycle, and the “spent 3 days in bed because I’m too depressed to do anything” thing really had to stop…

So after much research, I went shopping today and picked up the following supplements:

 

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Natural Balance “happy camper” herbal formula (one per day, in the morning)

A couple friends of mine swear by this stuff, so I figured I would try it.

Basically, it’s a proprietary blend of:
Passion Flower (aerial portion extract)
(helps relieve tension and complements the action of Kava Kava)

Kava Kava (root, rhizome)
(produces a pleasant, cheerful, and sociable feeling while helping to reduce anxiety)

Eleuthero (root)
(builds resistance to stress and fatigue)

Gotu Kola (aerial portion extract)
(calms the brain, enhances mental clarity, and helps the body adapt to stress)

Kola Nut (seed extract contains caffeine)
(cardiac and central-nervous-system stimulant)

Schizandra (fruit)
(brightens the mind )

Wood Betony (aerial portion)
(feeds and strengthens the nervous system)

Lavender (flower)
(removes nervous tension)

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and:

 

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NOW Mood Support With St. John’s Wort.  (one per day in the morning)

“Mood Support is a nutritional supplement that contains both nutrients and herbal extracts that aid in the support of a healthy nervous system and a positive, balanced mood state.”

Mood Support contains:
Thiamine (from Thiamine HCl) (Vitamin B-1) 4mg
Niacin (as Niacinamide) (Vitamin B-3) 25mg
Vitamin B-6 (from Pyridoxine HCl) 10mg
Folate (Folic Acid) 400mcg
Vitamin B-12 (as Cyanocobalamin) 200mcg
Pantothenic Acid (from D-Calcium Pantothenate) 50mg
Magnesium (from Magnesium Oxide and Amino Acid Chelate) 50mg
Zinc (from Zinc L-Methionine) 5mg
Manganese (from Manganese Amino Acid Chelate) 2mg
St. John’s Wort Extract (Hypericum perforatum) (Aerial Part with Flowers) (min. 0.3% Hypericin) 450mg
Holy Basil Extract (Tuisi Extract) (Leaves) (Ocimum tenuiflorum) (min. 2% Ursolic Acid) 100mg
Valerian 4:1 Extract (Valeriana officinalis) (Root) 50mg
5-HTP (5-hydroxytryptophan) (Griffonia simplicifolia) (Seed) 20mg
GABA (Gamma Aminobutyric Acid) (Free-Form) 250mg
Taurine (Free-Form) 125mg
L-Theanine (Suntheanine®) 50mg

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Note that this adds a substantial amount of B complex, but it is *very* hard to overdose on B complex, as they are all water soluble. This means excess vitamins are expelled in the urine. I don’t know anyone willing to take 30 high potency b complex tablets at once, but, that’s what it takes to “overdose” and the only result would be bright yellow urine. So, while you can od on B complex, its only dangerous in people who are already allergic to one of the B vitamins.

This works very well in conjunction with the “Happy Camper” mentioned above.

 

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NOW Melatonin (one per day, taken at bedtime)

Melatonin is a naturally occurring hormone produced by the pineal gland that plays a role in the regulation of the sleep-wake cycle. The pineal is embedded in the brain and receives signals via the optic nerve. Melatonin is secreted by the gland after the body senses darkness at the end of each day to keep our bodies synchronized with the rhythms of day and night. Melatonin is often used as a home remedy for jet lag.

Dietary tryptophan is converted to 5-HTP, which in turn can convert to serotonin and then to melatonin. People with diets low in tryptophan or who lack darkness at night may have a functional deficit of serotonin and melatonin, making melatonin supplementation useful.

Melatonin is also a powerful antioxidant that supports immune system function and is an antagonist to cortisol, the stress hormone produced by the adrenal glands. Cortisol is partly responsible for many of the classic symptoms of chronic stress, including lack of sleep.

Human melatonin production decreases as a person ages.

Light dependence
Production of melatonin by the pineal gland is inhibited by light and permitted by darkness. For this reason melatonin has been called “the hormone of darkness”. Its onset each evening is called the Dim-Light Melatonin Onset (DLMO). Secretion of melatonin as well as its level in the blood, peaks in the middle of the night, and gradually falls during the second half of the night, with normal variations in timing according to an individual’s chronotype. Terman et al. devised a formulation which mimics that gradual washout (vs. the spikes in blood concentration and rapid washout associated with most over-the-counter melatonin tablets). When used several hours before sleep, the compound shifts the circadian clock earlier, thus promoting earlier sleep onset and morning awakening.

Antioxidant
Besides its function as synchronizer of the biological clock, melatonin also exerts a powerful antioxidant activity. The discovery of melatonin as an antioxidant was made in 1993. In many less complex life forms, this is its only known purpose. Melatonin is an antioxidant that can easily cross cell membranes and the blood-brain barrier. Melatonin is a direct scavenger of OH, O2-, and NO. Unlike other antioxidants, melatonin does not undergo redox cycling, the ability of a molecule to undergo reduction and oxidation repeatedly. Redox cycling may allow other antioxidants (such as vitamin C) to act as pro-oxidants, counter intuitively promoting free radical formation. Melatonin, on the other hand, once oxidized, cannot be reduced to its former state because it forms several stable end-products upon reacting with free radicals. Therefore, it has been referred to as a terminal (or suicidal) antioxidant.

Recent research indicates that the first metabolite of melatonin in the melatonin antioxidant pathway may be N(1)-acetyl-N(2)-formyl-5-methoxykynuramine (or AFMK) rather than the common, excreted 6-hydroxymelatonin sulfate. AFMK alone is detectable in unicellular organisms and metazoans. A single AFMK molecule can neutralize up to 10 ROS/RNS (reactive oxygen species/reactive nitrogen species) since many of the products of the reaction/derivatives (including melatonin) are themselves antioxidants. This capacity to absorb free radicals extends at least to the quaternary metabolites of melatonin, a process referred to as “the free radical scavenging cascade”. This is not true of other, conventional antioxidants.

In animal models, melatonin has been demonstrated to prevent the damage to DNA by some carcinogens, stopping the mechanism by which they cause cancer. It also has been found to be effective in protecting against brain injury caused by ROS release in experimental hypoxic brain damage in newborn rats. Melatonin’s antioxidant activity may reduce damage caused by some types of Parkinson’s disease, may play a role in preventing cardiac arrhythmia and may increase longevity; it has been shown to increase the average life span of mice by 20% in some studies.

Immune system
While it is known that melatonin interacts with the immune system, the details of those interactions are unclear. There have been few trials designed to judge the effectiveness of melatonin in disease treatment. Most existing data are based on small, incomplete clinical trials. Any positive immunological effect is thought to result from melatonin acting on high affinity receptors (MT1 and MT2) expressed in immunocompetent cells. In preclinical studies, melatonin may enhance cytokine production, and by doing this counteract acquired immunodeficiences. Some studies also suggest that melatonin might be useful fighting infectious disease including viral, such as HIV, and bacterial infections, and potentially in the treatment of cancer.

Endogenous melatonin in human lymphocytes has been related to interleukin-2 (IL-2) production and to the expression of IL-2 receptor. This suggests that melatonin is involved in the clonal expansion of antigen-stimulated human T lymphocytes. When taken in conjunction with calcium, it is an immunostimulator and is used as an adjuvant in some clinical protocols; conversely, the increased immune system activity may aggravate autoimmune disorders. In rheumatoid arthritis patients, melatonin production has been found increased when compared to age-matched healthy controls.

Dreaming
Some supplemental melatonin users report an increase in vivid dreaming. Extremely high doses of melatonin (50 mg) dramatically increased REM sleep time and dream activity in both people with and without narcolepsy. Many psychoactive drugs, such as cannabis and lysergic acid diethylamide (LSD), increase melatonin synthesis. It has been suggested that nonpolar (lipid-soluble) indolic hallucinogenic drugs emulate melatonin activity in the awakened state and that both act on the same areas of the brain.

Autism
Individuals with autism spectrum disorders (ASD) may have lower than normal levels of melatonin. A 2008 study found that unaffected parents of individuals with ASD also have lower melatonin levels, and that the deficits were associated with low activity of the ASMT gene, which encodes the last enzyme of melatonin synthesis.

Current and potential medical indications
Melatonin has been studied for the treatment of cancer, immune disorders, cardiovascular diseases, depression, seasonal affective disorder (SAD), circadian rhythm sleep disorders and sexual dysfunction. Studies by Alfred J. Lewy at Oregon Health & Science University and other researchers have found that it may ameliorate circadian misalignment and SAD. Basic research indicates that melatonin may play a significant role in modulating the effects of drugs of abuse such as cocaine.

Treatment of circadian rhythm disorders
Exogenous melatonin taken in the evening is, together with light therapy upon awakening, the standard treatment for delayed sleep phase syndrome (DSPS) and non-24-hour sleep-wake syndrome. It appears to have some use against other circadian rhythm sleep disorders as well, such as jet lag and the problems of people who work rotating or night shifts. Melatonin reduces sleep onset latency to a greater extent in people with DSPS than in people with insomnia.

Taken 30 to 90 minutes before bedtime, melatonin supplementation acts as a mild hypnotic. It causes melatonin levels in the blood to rise earlier than the brain’s own production accomplishes. This usage is now commonly used in sleep and relaxation drinks.

A very small dose taken several hours before bedtime in accordance with the phase response curve for melatonin in humans (PRC) doesn’t cause sleepiness but, acting as a chronobiotic (affecting aspects of biological time structure), advances the phase slightly and is additive to the effect of using light therapy upon awakening. Light therapy may advance the phase about one to two-and-a-half hours and a small oral dose melatonin, timed correctly some hours before bedtime, can add about 30 minutes to the advance achieved with light therapy.

ADHD
Research shows that after melatonin is administered to ADHD patients on methylphenidate, the time needed to fall asleep is significantly reduced. Furthermore, the effects of the melatonin after three months showed no change from its effects after one week of use.

Mood disorders
Melatonin has been shown to be effective in treating one form of depression, seasonal affective disorder, and is being considered for bipolar and other disorders where circadian disturbances are involved. It has been observed that bipolar disorder might have, as a “trait marker” (something which is characteristic of being bipolar, that does not change with state), supersensitivity to light, i.e. a greater decrease in melatonin secretion in response to light exposure at night. This could be contrasted with drug-free recovered bipolar patients not showing light hypersensitivity.

Cancer
A systematic review of unblinded clinical trials involving a total of 643 cancer patients using melatonin found a reduced incidence of death. Another clinical trial is due to be completed in 2012. Melatonin levels at night are reduced to 50% by exposure to a low-level incandescent bulb for only 39 minutes, and it has been suspected that women with the brightest bedrooms have an increased risk for breast cancer. Reduced melatonin production has been proposed as a likely factor in the significantly higher cancer rates in night workers.

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Yeah, so it’s been working so far :)
-stone

Tuesday, June 14, 2011, 9:34 pm
So far so good.
Further research into melatonin shows that the recommended dosages are 1mg, and lower dosages are better.
Further research shows that it is very likely I am “D” deficient.
Further research shows that it is very likely I am “C” deficient.
I ran out of “Happy Camper” 2 weeks ago, and the effects were noticeable.
I ran out of “Mood Support” a week ago, and the effects were noticeable.

Health Rider Inversion Table System

Last week I whacked my back again, so my exercise routine has changed a bit, namely I now am doing 10 minutes of stretching on an inversion table, and then 10 side bends (each way) and 10 situps, while inverted, to help strengthen the area.

 

 

 

 

 

 

 

 

 

 

I’ve also upped my mileage to 5 per day, I’ll probably leave it here for a while.

Today I restocked my “Happy Camper”, “Mood Support” and Melatonin.
I’m changing the Melatonin dosage from 3mg to 1mg, taken nightly at bedtime.

I’m _not_ changing the Happy Camper, Mood support, or Omega 3 dosages at this time.

I also made some other additions to my supplement routine.

 

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Spring Valley Natural C with rose hips 500mg (one per day, in the morning)

You know, to prevent Scurvy :)

 

 

 

 

 

 

 

 

 

 

 

 

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and:

 

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Spring Valley Maximum Strength D3 5000 iu (one per day, in the morning)

Bone health

One of the most important roles of vitamin D is to maintain skeletal calcium balance by promoting calcium absorption in the intestines, promoting bone resorption by increasing osteoclast number, maintaining calcium and phosphate levels for bone formation, and allowing proper functioning of parathyroid hormone to maintain serum calcium levels.

Vitamin D deficiency can result in lower bone mineral density and an increased risk of bone loss (osteoporosis) or bone fracture because a lack of vitamin D alters mineral metabolism in the body.

Vitamin D has been studied as a potential treatment for osteoporosis, but since treatment of vitamin D deficiency is associated with an increase of mineralization of osteoid, it remains unclear whether vitamin D has any effect on osteoporotic bone.

In cross-sectional studies there was a positive relationship between vitamin D and bone mineral density in the hip.

Lips (2001) reported that bone mineral deficit in osteomalacia was larger than that in milder degrees of vitamin D deficiency.

There is also a relationship between low bone mineral density and sedentary life style. This is evident in frail, elderly subjects because they are often vitamin D deficient and lead an inactive lifestyle.

Lips (2001) also reported that mild vitamin D deficiency was not associated with an increased risk for hip fracture. A study done in Norway consisted of 246 patients with hip fractures who were studied for risk factors.

Results showed that a vitamin D intake lower than 100 IU/day was associated with an increased risk for hip fracture.

Vitamin D supplements may also increase bone mineral density in other parts of the skeleton.

A study showed that a supplement of 800 IU per day of vitamin D increased the bone mineral density of the lumbar spine in postmenopausal women in comparison with the control group.

Persons over the age of 50 years need higher levels of vitamin D. In a study discussed in LoPiccolo et al. (2010), adults who consumed a daily supplementation with 482–770 IU of vitamin D had reduced fracture rates of 20% for non-vertebral fractures.  However, there was no reported reduction in fracture risk for persons who had 400 IU or less of vitamin D daily.

 

Immune system

Vitamin D receptor ligands have been shown to increase the activity of natural killer cells, and enhance the phagocytic activity of macrophages.

Active vitamin D hormone also increases the production of cathelicidin, an antimicrobial peptide that is produced in macrophages triggered by bacteria, viruses, and fungi.

Suggestions of a link between vitamin D deficiency and the onset of multiple sclerosis posited that this is due to the immune-response suppression properties of Vitamin D and that vitamin D is required to activate a histocompatibility gene (HLA-DRB1*1501) necessary for differentiating between self and foreign proteins in a subgroup of individuals genetically predisposed to MS.

Whether vitamin D supplements during pregnancy can lessen the likelihood of the child developing MS later in life is not known; however, vitamin D fortification has been suggested to have caused a pandemic of allergic disease and an association between vitamin D supplementation in infancy and an increased risk of atopy and allergic rhinitis later in life has been found. Veteran vitamin D researcher Hector DeLuca has cast doubt on whether vitamin D affects MS.
Tuberculosis and HIV

Historically, vitamin D3 was used to treat tuberculosis patients, but has not been adequately investigated in controlled clinical trials. The hormonally active form of vitamin D3, 1,25-dihydroxycholecalciferol (1,25(OH)2D), has been shown to have antimycobacterial activity in vitro, but the applicability of this effect to clinical situations has not been shown. Vitamin D3 supplementations has not shown any improvement in treating tuberculosis except in a small subset of patients with the tt genotype of the TaqI vitamin D receptor polymorphism. Several studies have shown an association between low serum levels of 25-hydroxycholecalciferol (25(OH)D) and increased risk for both active tuberculosis disease progression and susceptibility. More prospective studies will be required to ascertain the potential role of vitamin D supplementation in treating patients with tuberculosis.

Vitamin D3 has also shown some anti-HIV-1 effects in vitro, including the induction of autophagy. The potential effect in humans has not been investigated. Lower levels of 1,25(OH)2D in HIV infected patients are correlated with significantly lower CD4+ T cell counts and higher tumor necrosis factor levels, which normally decrease in number with progression to AIDS, although no causative association has been shown. In an epidemiological study of HIV positive women in Tanzania, there appeared to be a correlation between reduced levels of Vitamin D and speed of HIV disease progression. These results will need to be confirmed in a blinded clinical trial before dietary recommendations can be made.

Influenza
See also: Vitamin D and influenza

Lack of vitamin D synthesis is a possible explanation for high rates of influenza infection during winter however, see flu season for the factors apart from vitamin D that are also hypothesized to influence rates of infection during winter. For viral infections, other implicated factors include low relative humidities produced by indoor heating and cold temperatures that favor virus spread during winter.

Cancer

The molecular basis for thinking that vitamin D has the potential to prevent cancer lies in its role in a wide range of cellular mechanisms central to the development of cancer. These effects may be mediated through vitamin D receptors expressed in cancer cells. Polymorphisms of the vitamin D receptor (VDR) gene have been associated with an increased risk of breast cancer. Women with mutations in the VDR gene had an increased risk of breast cancer.

A 2006 study using data on over 4 million cancer patients from 13 different countries showed a marked increase in some cancer risks in countries with less sun and another metastudy found correlations between vitamin D levels and cancer. The authors suggested that intake of an additional 1,000 international units (IU) (or 25 micrograms) of vitamin D daily reduced an individual’s colon cancer risk by 50%, and breast and ovarian cancer risks by 30%. Low levels of vitamin D in serum have been correlated with breast cancer disease progression and bone metastases. However, the vitamin D levels of a population do not depend on the solar irradiance to which they are exposed. Moreover, there are genetic factors involved with cancer incidence and mortality which are more common in northern latitudes.

A 2006 study found that taking the U.S. RDA of vitamin D (400 IU per day) cut the risk of pancreatic cancer by 43% in a sample of more than 120,000 people from two long-term health surveys. However, in male smokers a 3-fold increased risk for pancreatic cancer in the highest compared to lowest quintile of serum 25-hydroxyvitamin D concentration has been found.

A randomized intervention study involving 1,200 women, published in June 2007, reports that vitamin D supplementation (1,100 international units (IU)/day) resulted in a 60% reduction in cancer incidence, during a four-year clinical trial, rising to a 77% reduction for cancers diagnosed after the first year (and therefore excluding those cancers more likely to have originated prior to the vitamin D intervention). The study was criticized on several grounds including lack of reported data, use of statistical techniques and comparison with a self-selected (i.e. non-randomized) observational study that found long term convergence of breast cancer incidence (i.e. the cancer occurrence had merely been delayed)  The author’s response provided the requested data, explained their statistical usage and commented that even if the vitamin D merely delayed the appearance of cancer (which they did not believe, based on other studies), that this was still a considerable benefit.

In 2007, the Canadian Cancer Society recommended that adults living in Canada should consider taking vitamin D supplementation of 1,000 international units (IU) a day during the fall and winter. A US National Cancer Institute study analyzed data from the third national Health and Nutrition Examination Survey to examine the relationship between levels of circulating vitamin D in the blood and cancer mortality in a group of 16,818 participants aged 17 and older. It found no support for an association between 25(OH)D and total cancer mortality. However, the study did find that “colorectal cancer mortality was inversely related to serum 25(OH)D level, with levels 80 nmol/L or higher associated with a 72% risk reduction (95% confidence interval = 32% to 89%) compared with lower than 50 nmol/L, Ptrend = .02.” Unlike other studies, this one was carried out prospectively — meaning that participants were followed looking forward — and the researchers used actual blood tests to measure the amount of vitamin D in blood, rather than trying to infer vitamin D levels from potentially inaccurate predictive models.

A meta-study published in the International Journal of Cancer in May 2010 analyzed 35 independent studies of vitamin D and cancer. The researchers determined that a 10 nanogram/milliliter increase in serum vitamin D is associated with a 15% lower risk of colon cancer. The analysis also found an 11% lower risk for breast cancer, although the authors report that due to case study methodology that this finding is ultimately insignificant.

A 2011 study done at the University of Rochester Medical Center found that low vitamin D levels among women with breast cancer correlate with more aggressive tumors and poorer prognosis. The study associated sub-optimal vitamin D levels with poor scores on every major biological marker that helps physicians predict a patient’s breast cancer outcome. The lead researcher stated, “Based on these results, doctors should strongly consider monitoring vitamin D levels among breast cancer patients and correcting them as needed.”

Cardiovascular disease

A report from the National Health and Nutrition Examination Survey (NHANES) involving nearly 5,000 participants found that low levels of vitamin D were associated with an increased risk of peripheral artery disease (PAD). The incidence of PAD was 80% higher in participants with the lowest vitamin D levels (<17.8 ng/mL). Cholesterol levels were found to be reduced in gardeners in the UK during the summer months. Low levels of vitamin D are associated with an increase in high blood pressure and cardiovascular risk. Numerous observational studies show this link, but of two systemic reviews one found only weak evidence of benefit from supplements and the other found no evidence of a beneficial effect whatsoever.

There is a certain amount of evidence to suggest that dietary vitamin D may be carried by lipoprotein particles into cells of the artery wall and atherosclerotic plaque, where it may be converted to active form by monocyte-macrophages. These findings raise questions regarding the effects of vitamin D intake on atherosclerotic calcification and cardiovascular risk. Calcifediol (25-hydroxy-vitamin D) is implicated in the etiology of atherosclerosis, especially in non-Caucasians. Freedman et al. (2010) found that serum vitamin D correlates with calcified atheroscleratic plaque (CP) in African Americans, but not in Euro-Americans, “Higher levels of 25-hydroxyvitamin D seem to be positively associated with aorta and carotid CP in African Americans but not with coronary CP. These results contradict what is observed in individuals of European descent.” One study found an elevated risk of ischaemic heart disease in Southern India in individuals whose vitamin D levels were above 89 ng/mL. A review of vitamin D status in India concluded that studies uniformly point to low 25(OH)D levels in Indians despite abundant sunshine, and suggested a public health need to fortify Indian foods with vitamin D might exist. The levels found in India are consistent with many other studies of tropical populations which have found that even an extreme amount of sun exposure, such as incurred by rural Indians, does not raise 25(OH)D levels to the levels typically found in Europeans.

Mortality

Using information from the National Health and Nutrition Examination Survey a large scale study concluded that having low levels of vitamin D (<17.8 ng/ml) was independently associated with an increase in all-cause mortality in the general population. However it has been pointed out that increased mortality was also found in those with higher concentrations, (above 50 ng/ml). A sophisticated August 2010 study of plasma vitamin D and mortality in older men concluded that both high (>39 ng/ml) and low (<18 ng/ml)) concentrations of plasma 25(OH)D are associated with elevated risks of overall and cancer mortality compared with intermediate concentrations. These boundaries were less than suggested by the Melamed et al. study of National Health and Nutrition Examination Survey data but the immunoassay used by National Health and Nutrition Examination Survey tended to overestimate vitamin D values.

Overall, excess or deficiency in the calciferol system appear to cause abnormal functioning and premature aging.

Complex regulatory mechanisms control metabolism and recent epidemiological evidence suggests that there is a narrow range of vitamin D blood levels in which metabolic functions are optimized. Levels above or below this natural homeostasis of vitamin D are associated with increased mortality.

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and:

 

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NOW 5-HTP “neurotransmitter support” 50mg (one per day, in the evening)
Note that this is taking the 20mg of 5-HTP in “Mood Support” up to 70mg.

5-HTP has been studied and shown to be of benefit in the following conditions: depression, anxiety, binge eating associated with obesity, and insomnia.
Based on the occurrence of serious side effects and drug interactions (q.v.), particularly in long-term use in form of dietary supplements that are often not communicated to the treating physician, its use is no longer recommended. In addition, due to unspecific effects, the risk/benefit ratio compared to serotonin reuptake inhibitors and other serotonergic antidepressant drugs is not favorable for 5-HTP containing supplements.

I’m going to walk softly with this, and if i don’t see any changes in a week I will probably stop taking it, as things like:

“Direct and indirect evidence for possible yet unproven risks and side effects associated with 5-HTP when overdosed: Heart valve damage or disease (cardiac fibrosis). When combined with MAOIs or SSRIs, 5-HTP can cause acute serotonin syndrome.”

Are warnings I do not take lightly.

I feel comfortable with this at this time, as it is widely agreed upon that excess 5-HTP, especially when administered with Vitamin B6, is thought to be metabolized and excreted, and I’m taking *a lot* of  B6.

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I’m also looking into:

 

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NOW L-Tryptophan 500mg (one per day, before bedtime)
For many organisms (including humans), tryptophan is an essential amino acid. This means that it cannot be synthesized by the organism and therefore must be part of its diet. Amino acids, including tryptophan, act as building blocks in protein biosynthesis. In addition, tryptophan functions as a biochemical precursor for the following compounds (see also figure to the right):

Serotonin (a neurotransmitter), synthesized via tryptophan hydroxylase. Serotonin, in turn, can be converted to melatonin (a neurohormone), via N-acetyltransferase and 5-hydroxyindole-O-methyltransferase activities.

Niacin is synthesized from tryptophan via kynurenine and quinolinic acids as key biosynthetic intermediates.

Auxin (a phytohormone) when sieve tube elements undergo apoptosis tryptophan is converted to auxins.

The disorders fructose malabsorption and lactose intolerance cause improper absorption of tryptophan in the intestine, reduced levels of tryptophan in the blood and depression.

In bacteria that synthesize tryptophan, high cellular levels of this amino acid activate a repressor protein, which binds to the trp operon. Binding of this repressor to the tryptophan operon prevents transcription of downstream DNA that codes for the enzymes involved in the biosynthesis of tryptophan. So high levels of tryptophan prevent tryptophan synthesis through a negative feedback loop and, when the cell’s tryptophan levels are reduced, transcription from the trp operon resumes. The genetic organisation of the trp operon thus permits tightly regulated and rapid responses to changes in the cell’s internal and external tryptophan levels.

Dietary sources
Tryptophan is a routine constituent of most protein-based foods or dietary proteins. It is particularly plentiful in chocolate, oats, dried dates, milk, yogurt, cottage cheese, red meat, eggs, fish, poultry, sesame, chickpeas, sunflower seeds, pumpkin seeds, spirulina, and peanuts. Despite popular belief that turkey has a particularly high amount of tryptophan, the amount of tryptophan in turkey is typical of most poultry.

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Since other than cheese, and the occasional ice cream, I avoid all dairy, and I have avoided all nuts for years,  (although one would think that my intake of red meat, eggs, fish, and poultry would be enough).

To take this one,  It looks like I may have to drop the Mood Support, due to the potential for serotonin overdose when combined with St Johns Wort.

so far, so good :)

-stone

custom rom your htc hero…

ahh..
so, my sprint htc hero and i have seen many miles together, but as i use it for more and more things, it was getting kinda sluggish…
.. and somadina’s was so bad she had sprint send a replacement, and it’s just as bad.

droid does indeed.

so i did some digging, and found my answers…

fresh.

custom roms are everywhere for this phone, and it seems the primary customer complaint about the phone is that it’s slow, and sluggish, and there’s a bunch of sprint applications that auto start and run in the background that most people will never use…

shame on sprint for making it so you have to hack root to uninstall any of it….

enter flipz and his posse of 31337 haxors, and the fresh rom.

when i saw the list of what was removed…

amazon mp3
gsd
htc footprints and widget
htc mobile guide (aka learn more)
htc navigation panel
htc ringtone trimmer
htc stocks and widget
google im
quick office
sdsa (sprint self service)
sprint nascar
sprint navigation
sprint nfl
teeter

i smiled, because i wanted all of that gone.

then i looked at the list of stuff they added in…

had me hooked at “wifi tether”

so here we go…

make a backup
(instructions are [here])

root your phone.
(instructions are [here] (android 1.5) and [here] (android 2.x))

grab the fresh rom (i’m running 2.4.0) from [here] and copy it to your sdcard.

open your command prompt window, go to c:\droidsdk\tools

type adb shell and hit [enter]
type reboot recovery and hit [enter]

select wipe data/factory reset.
select wipe cache partition.
select install zip from sdcard.
select fresh rom from sdcard.

once it’s done, select “go back” to get to the main menu, and reboot system now.

you should reboot into fresh.

be patient, it may take a while.
if you get stuck in a reboot loop, you forgot the “wipe” step.

when the phone comes back up, set your google up right away, so it can sync your contacts while you’re messing with the rest of it.

by the way, flipz removed sprint nav.
before you reinstall it, you should check out google maps / navigation.
i found it to be much much faster and way more accurate.

enjoy :)
-stone.

root your sprint htc hero (android 2.x)…

note: these are the instructions for rooting android 2.x. android 1.5 rooting instructions are [here]

so they forced a patch that plugged the root hole in android 2.1, so if you want to flash a custom rom,
you have to jump through a few more hoops…

start by grabbing the pre patch rom [here]

if you don’t have it yet, grab htc sync from [here] and install it

plug your phone into your pc via usb cable.

run the pre patch rom executable.

follow the on screen instructions until you reboot.

when the phone comes back up, it’s going to ask you about an update. tell it *no*, or you’re back where you started.

the rest of this operation will require the android SDK (software development kit).
you can download it [here]

extract the files to c:\droidsdk

save yourself some pain and after you extract the files, make sure your tree looks like:

c:\droidsdk\tools (and all the other folders) because it doesn’t unpack that way sometimes.

on your phone, click menu > settings > applications > development and make sure usb debugging is turned on.

plug your phone into your computer via usb. make sure that the connection type in the notification pull down is “charge only”.

it should install the drivers automagically.

once you have the SDK installed, open a command prompt (click start, click run, type cmd, hit [enter])

type cd \droidsdk\tools\ and hit [enter]
type adb devices and hit [enter]

it should reply with the serial number of your phone.

if it doesn’t, reinstall the drivers.

once you get the proper response from your command prompt, grab the root kit [here] and unzip it to c:\droidsdk\tools\

in your command prompt window:
adb push unrevoked /sdcard and hit [enter]
adb push recovery.img /sdcard and hit [enter]
adb push boot-patched.zip /sdcard and hit [enter]

now we root it:

type adb shell and hit [enter]

it should reply with $

type sh /sdcard/unrevoked and hit [enter]

it should give you a screen full of ascii text and a # prompt.

go to your apps menu on your phone, you should see a “superuser” application.
run it, you should see a blank screen.

on your computer, in the command prompt window, type su and hit [enter]
a prompt will appear on your hero asking you to give /system/bin/sh superuser access. tell it yes.

your command prompt should say:

$ su
su
#

now type flash_image recovery /sdcard/recovery.img and hit [enter]

your command prompt should say:

# flash_image recovery /sdcard/recovery.img
flash_image recovery /sdcard/recovery.img
#

type reboot recovery and hit [enter]

your phone will now reboot into the recovery image you just flashed to it.

in the recovery menu on your phone, choose “flash zip from sdcard”

then select “boot-patched.zip”

and press the home button to apply the changes.

once Recovery says “install from sdcard complete”, you should probably make a nandroid backup…

in the recovery menu, select “backup/restore” and then select “nand backup”.

it saves the backup in /sdcard/nandroid/(your serial number)/

you need to disable the ota updates or you will lose root on the next update.

that is done in your command prompt.

type cd C:\droidsdk\tools and hit [enter]
type adb remount and hit [enter]
type adb shell and hit [enter]
type mv /system/etc/security/otacerts.zip /system/etc/security/otacerts.zip.bak and hit [enter]

this does not prevent the updates from downloading, but they will fail their signature checks and will not install.

welcome to root :)
-stone

a moment of silence…

as my 42 minutes of silence (8:46 am – 10:28 am september 11) comes to a close, i am thankful for all of those in harms way… the ones who’s blood forms the thin red line that holds the evil of the world in check as best they can.

thank you.
-stone

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